추천 제품
Quality Level
분석
≥98% (HPLC)
양식
powder
색상
white to beige
solubility
DMSO: 5 mg/mL (clear solution)
저장 온도
2-8°C
SMILES string
O=C1N(c2ccccc2)[Se](=O)c3ccccc13
InChI
1S/C13H9NO2Se/c15-13-11-8-4-5-9-12(11)17(16)14(13)10-6-2-1-3-7-10/h1-9H
InChI key
SBTLFLABILGUMK-UHFFFAOYSA-N
생화학적/생리학적 작용
Ebselen Oxide is an oxidative product of ebselen containing selenoxide group. Unlike ebselen, it lacks anti-oxidative property. Ebselen oxide may suppress human immunodeficiency virus-1 (HIV-1) replication by inhibiting HIV-1 capsid protein, similar to ebselen.
Ebselen oxide is a potent inhibitor of α-Methylacyl coenzyme A racemase (AMACR).
Ebselen oxide is a potent inhibitor of a-Methylacyl coenzyme A racemase (AMACR).
신호어
Danger
유해 및 위험 성명서
Hazard Classifications
Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Skin Irrit. 2 - STOT RE 2 - STOT SE 3
표적 기관
Respiratory system
Storage Class Code
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
WGK
WGK 3
Flash Point (°F)
Not applicable
Flash Point (°C)
Not applicable
가장 최신 버전 중 하나를 선택하세요:
Yuren Wang et al.
Drug design, development and therapy, 11, 1369-1382 (2017-05-13)
Histone deacetylases (HDACs) are key regulators of gene expression in cells and have been investigated as important therapeutic targets for cancer and other diseases. Different subtypes of HDACs appear to play disparate roles in the cells and are associated with
Chien-Feng Li et al.
Clinical cancer research : an official journal of the American Association for Cancer Research, 20(23), 6141-6152 (2014-11-12)
Myxofibrosarcomas frequently display arm-level gains on 5p. We characterized the pathogenetic and therapeutic relevance of the α-methylacyl coenzyme A racemase (AMACR) at 5p13.3. AMACR mRNA expression in myxofibrosarcomas was analyzed using the public transcriptome and laser-microdissected sarcoma cells. We performed
Chien-Feng Li et al.
Oncotarget, 5(22), 11588-11603 (2014-12-05)
Non-random gains of chromosome 5p have been observed in clinically aggressive gastrointestinal stromal tumors, whereas the driving oncogenes on 5p remain to be characterized. We used an integrative genomic and functional approach to identify amplified oncogenes on 5p and to
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