MAB5488
Anti-CRABP2 Antibody
ascites fluid, Chemicon®
Synonym(s):
CRABPII
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About This Item
UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41
Recommended Products
biological source
mouse
Quality Level
antibody form
ascites fluid
antibody product type
primary antibodies
clone
monoclonal
species reactivity
mouse, human
manufacturer/tradename
Chemicon®
technique(s)
ELISA: suitable
immunocytochemistry: suitable
western blot: suitable
isotype
IgG2a
NCBI accession no.
Specificity
Reacts with Cellular Retinoic Acid Binding Protein II (CRABPII). No cross reactivity with CRABPI.
Immunogen
Full length recombinant human CRABPII.
Application
Use Anti-CRABP2 Antibody (Mouse Monoclonal Antibody) validated in ELISA, WB, ICC to detect CRABP2 also known as Cellular Retinoic Acid Binding Protein II.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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Storage Class
10 - Combustible liquids
wgk_germany
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
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Temporal blastemal cell gene expression analysis in the kidney reveals new Wnt and related signaling pathway genes to be essential for Wilms' tumor onset.
Maschietto, M; Trape, AP; Piccoli, FS; Ricca, TI; Dias, AA; Coudry, RA; Galante et al.
Cell Death & Disease null
L Delva et al.
Molecular and cellular biology, 19(10), 7158-7167 (1999-09-22)
Two sorts of proteins bind to, and mediate the developmental and homeostatic effects of, retinoic acid (RA): the RAR and RXR nuclear receptors, which act as ligand-dependent transcriptional regulators, and the cellular RA binding proteins (CRABPI and CRABPII). CRABPs are
Cutaneous clear cell squamous cell carcinoma in situ : clinical, histological and immunohistochemical characterization.
Munir Yahya Hussein Al-Arashi,H Randolph Byers
Journal of cutaneous pathology null
Epigenetic silencing of CRABP2 and MX1 in head and neck tumors.
Calmon MF, Rodrigues RV, Kaneto CM, Moura RP, Silva SD, Mota LD, Pinheiro DG, Torres C et al.
Neoplasia null
Shuiliang Yu et al.
Journal of experimental & clinical cancer research : CR, 41(1), 88-88 (2022-03-10)
Resistance to standard therapy is a major reason for the poor prognosis of pancreatic ductal adenocarcinoma (PDAC). Developing novel therapy to overcome PDAC drug-resistance is urgently needed. CRABP-II was highly expressed in all PDAC but not expressed in normal pancreatic
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